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Kevin S. W. Tan

Researcher at National University of Singapore

Publications -  147
Citations -  10493

Kevin S. W. Tan is an academic researcher from National University of Singapore. The author has contributed to research in topics: Blastocystis & Plasmodium falciparum. The author has an hindex of 38, co-authored 124 publications receiving 8614 citations. Previous affiliations of Kevin S. W. Tan include Tan Tock Seng Hospital & Rockefeller University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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New insights on classification, identification, and clinical relevance of Blastocystis spp.

Kevin S. W. Tan
- 01 Oct 2008 - 
TL;DR: Accumulating epidemiological, in vivo, and in vitro data strongly suggest that Blastocystis is a pathogen, and a suggestion that previously conflicting observations on the pathogenesis of BlastocyStis are due to pathogenic and nonpathogenic genotypes is suggested.
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Haem-activated promiscuous targeting of artemisinin in Plasmodium falciparum

Jigang Wang, +19 more
- 22 Dec 2015 - 
TL;DR: It is shown that haem, rather than free ferrous iron, is predominantly responsible for artemisinin activation, which derives primarily from the parasite's haem biosynthesis pathway at the early ring stage and from haemoglobin digestion at the latter stages.
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Terminology for Blastocystis subtypes – a consensus

C. Rune Stensvold, +7 more
- 01 Mar 2007 - 
TL;DR: It is proposed that all mammalian and avian isolates should be designated Blastocystis sp.
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Deformability based cell margination—A simple microfluidic design for malaria-infected erythrocyte separation

Han Wei Hou, +8 more
- 06 Sep 2010 - 
TL;DR: It is demonstrated using microfluidics that stiffer malaria-infected RBCs (iRBCs) behave similar to leukocytes and undergo margination towards the sidewalls, which provides better understanding of the hemodynamic effects of iR BCs in microcirculation and its contribution to pathophysiological outcome relating to cytoadherence to endothelium.