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Terry D. Oberley

Researcher at University of Wisconsin-Madison

Publications -  186
Citations -  18249

Terry D. Oberley is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Superoxide dismutase & Oxidative stress. The author has an hindex of 67, co-authored 186 publications receiving 16852 citations. Previous affiliations of Terry D. Oberley include Veterans Health Administration & Memorial Hospital of South Bend.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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CuZnSOD deficiency leads to persistent and widespread oxidative damage and hepatocarcinogenesis later in life

TL;DR: Mice deficient in CuZn superoxide dismutase showed no overt abnormalities during development and early adulthood, but had a reduced lifespan and increased incidence of neoplastic changes in the liver, which likely contributes to hepatocarcinogenesis later in life.
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The protective role of manganese superoxide dismutase against adriamycin-induced acute cardiac toxicity in transgenic mice.

TL;DR: Results support a major role for free radical generation in ADR toxicity as well as suggesting mitochondria as the critical site of cardiac injury.
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Comparison of the Pro-Oxidative and Proinflammatory Effects of Organic Diesel Exhaust Particle Chemicals in Bronchial Epithelial Cells and Macrophages

TL;DR: It is demonstrated that epithelial cells exhibit a hierarchical oxidative stress response that differs from that of macrophages by more rapid transition from cytoprotective to cytotoxic responses, and not being protected by a thiol antioxidant, N-acetylcysteine, which effectively protects macrophage against cytot toxic DEP chemicals.