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Federica Sotgia

Researcher at University of Salford

Publications -  250
Citations -  33056

Federica Sotgia is an academic researcher from University of Salford. The author has contributed to research in topics: Cancer cell & Stromal cell. The author has an hindex of 85, co-authored 247 publications receiving 28751 citations. Previous affiliations of Federica Sotgia include Manchester Academic Health Science Centre & The Breast Cancer Research Foundation.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The reverse Warburg effect: Aerobic glycolysis in cancer associated fibroblasts and the tumor stroma

Stephanos Pavlides, +13 more
- 01 Dec 2009 - 
TL;DR: In this alternative model of tumorigenesis, the epithelial cancer cells instruct the normal stroma to transform into a wound-healing stroma, providing the necessary energy-rich micro-environment for facilitating tumor growth and angiogenesis, explaining its powerful predictive value.
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Cancer metabolism: a therapeutic perspective

Ubaldo E. Martinez-Outschoorn, +5 more
- 01 Jan 2017 - 
TL;DR: How cancer cells reprogramme their metabolism and that of other cells within the tumour microenvironment in order to survive and propagate, thus driving disease progression is discussed; in particular, potential metabolic vulnerabilities that might be targeted therapeutically are highlighted.
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Mutations in the caveolin-3 gene cause autosomal dominant limb-girdle muscular dystrophy.

Carlo Minetti, +16 more
- 01 Apr 1998 - 
TL;DR: Eight patients from two different families are described with a new form of autosomal dominant LGMD, which is proposed to call LGMD1C, associated with a severe deficiency of caveolin-3 in muscle fibres, and two mutations in the gene are identified that may interfere with caveolae formation at the muscle cell plasma membrane.