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Linda F. van Dyk

Researcher at University of Colorado Denver

Publications -  49
Citations -  7488

Linda F. van Dyk is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Viral replication & Lytic cycle. The author has an hindex of 23, co-authored 47 publications receiving 6366 citations. Previous affiliations of Linda F. van Dyk include Washington University in St. Louis & Anschutz Medical Campus.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Identification of microRNAs of the herpesvirus family

Sébastien Pfeffer, +19 more
- 16 Feb 2005 - 
TL;DR: To identify other miRNA genes in pathogenic viruses, a new miRNA gene prediction method with small-RNA cloning from several virus-infected cell types was combined and predicted miRNAs in several large DNA viruses.
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T-box transcription factor T-bet, a key player in a unique type of B-cell activation essential for effective viral clearance

Kira Rubtsova, +5 more
- 20 Aug 2013 - 
TL;DR: A unique pathway of B- cell activation, leading to IgG2a production, and involving synergistic stimulation via B-cell antigen receptors, toll-like receptor 7 (TLR7), and IFNγ receptors on B cells is demonstrated, demonstrating a previously unknown role for T-bet expression in B cells during viral infections.
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The Murine Gammaherpesvirus 68 v-Cyclin Is a Critical Regulator of Reactivation from Latency

Linda F. van Dyk, +2 more
- 15 Aug 2000 - 
TL;DR: It is demonstrated that despite expression as a lytic cycle gene, the pro-proliferative γHV68 v-cyclin is not required for γ HV68 replication either in vitro or during acute infection in vivo but rather is a critical determinant of reactivation from latency.
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TCR antigen-induced cell death occurs from a late G1 phase cell cycle check point

Natalie A. Lissy, +5 more
- 01 Jan 1998 - 
TL;DR: Analysis of the timing of death, cell cycle markers, BrdU-labeled S phase cells, and phase-specific centrifugally elutriated cultures from stimulated Jurkat T cells and peripheral blood lymphocytes suggest that AID occurs from a late G1 death check point in a pRb-dependent fashion.