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Allen Kaasik

Researcher at University of Tartu

Publications -  62
Citations -  13207

Allen Kaasik is an academic researcher from University of Tartu. The author has contributed to research in topics: Mitochondrion & Mitophagy. The author has an hindex of 29, co-authored 61 publications receiving 10649 citations. Previous affiliations of Allen Kaasik include French Institute of Health and Medical Research & University of Paris-Sud.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

PGC-1{alpha} and PGC-1{beta} regulate mitochondrial density in neurons.

Przemyslaw Wareski, +6 more
- 07 Aug 2009 - 
TL;DR: Activation or overexpression of the P GC-1 family of coactivators could be used to compensate for neuronal mitochondrial loss and suggest that therapeutic agents activating PGC-1 would be valuable for treating neurodegenerative diseases in which mitochondrial dysfunction and oxidative damage play an important pathogenic role.
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Mutant A53T α-Synuclein Induces Neuronal Death by Increasing Mitochondrial Autophagy

Vinay Choubey, +7 more
- 25 Mar 2011 - 
TL;DR: Evidence is provided that the overactivation of autophagy may be a link that connects the intracellular accumulation of α-synuclein with mitochondrial dysfunction, and that overactivated mitochondrial removal could be one of the contributing factors that leads to the mitochondrial loss observed in PD models.