Chiara Di Pietro

TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.

TL;DR: The specific role of GPR37, a putative peptidergic G protein-coupled receptor, in modulating the functional expression of DAT and the behavioral responses to dopaminergic drugs is revealed.

TL;DR: It is shown that the ablation of the murine Gpr37l1 gene results in premature down-regulation of proliferation of granule neuron precursors and precocious maturation of Bergmann glia and Purkinje neurons.

TL;DR: Findings show that G PR37 overexpression per se can induce cellular autophagy, which may prevent the selective degeneration of GPR37‐expressing neurons, as reported for Parkinson's and related neurodegenerative diseases.

TL;DR: The lack of the GPR37/PAEL receptor affects both ERK2‐ and Akt‐mediated striatal signaling pathways, impairing the biochemical and behavioral responses typically induced by acute and chronic administration of psychostimulant drugs.