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Vladimir P. Skulachev

Researcher at Moscow State University

Publications -  464
Citations -  29741

Vladimir P. Skulachev is an academic researcher from Moscow State University. The author has contributed to research in topics: Mitochondrion & Respiratory chain. The author has an hindex of 78, co-authored 460 publications receiving 27163 citations. Previous affiliations of Vladimir P. Skulachev include University of Milan & University of Osnabrück.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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High protonic potential actuates a mechanism of production of reactive oxygen species in mitochondria.

Sergey S Korshunov, +2 more
- 13 Oct 1997 - 
TL;DR: Inhibition of H2O2 formation by uncoupler, malonate and ADP+Pi is shown to be proportional to the ΔΨ decrease by these compounds, which is in line with the concept that a high proton motive force in state 4 is potentially dangerous for the cell due to an increase in the probability of superoxide formation.
Journal ArticleDOI

Uncoupling: new approaches to an old problem of bioenergetics

Vladimir P. Skulachev
- 25 Feb 1998 - 
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Role of uncoupled and non-coupled oxidations in maintenance of safely low levels of oxygen and its one-electron reductants

Vladimir P. Skulachev
- 01 May 1996 - 
TL;DR: In this article, a concept is put forward that non-phosphorylating (uncoupled or non-coupled) respiration takes part in maintenance of low levels of both O2 and the O2 reductants when the normal respiration fails to do this job due to lack of ADP.
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Cytochrome c in the apoptotic and antioxidant cascades

Vladimir P. Skulachev
- 27 Feb 1998 - 
TL;DR: The role of cytochrome c in apoptosis may be understood within the framework of the concept assuming that the evolutionary primary function of apoptosis was to purify tissues from ROS‐overproducing cells and the pro‐apoptosis activity might represent one of the anti‐oxidant functions inherent in this cy tochrome.