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Stéphen Manon

Researcher at University of Bordeaux

Publications -  106
Citations -  10072

Stéphen Manon is an academic researcher from University of Bordeaux. The author has contributed to research in topics: Mitochondrion & Apoptosis. The author has an hindex of 40, co-authored 98 publications receiving 8715 citations. Previous affiliations of Stéphen Manon include Université Bordeaux Segalen & Centre national de la recherche scientifique.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Uth1p is involved in the autophagic degradation of mitochondria

Ingrid Kiššová, +3 more
- 10 Sep 2004 - 
TL;DR: Data show that, although the autophagic machinery was fully functional in the absence of Uth1p, this protein is involved in theAutophagic degradation of mitochondria.
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Bax activation and mitochondrial insertion during apoptosis.

Lisenn Lalier, +9 more
- 15 Feb 2007 - 
TL;DR: This review focuses on the molecular events of Bax activation through its interaction with the other proteins from the Bcl-2 family, and the mechanism by which Bax triggers the MOM permeabilization once activated.
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Release of cytochrome c and decrease of cytochrome c oxidase in Bax-expressing yeast cells, and prevention of these effects by coexpression of Bcl-xL

Stéphen Manon, +2 more
- 22 Sep 1997 - 
TL;DR: It is found that Bax‐induced growth arrest of yeast cells is related to two defects in the respiratory chain: a decrease in the amount of cytochrome c oxidase and a dramatic increase in the release of cy tochrome c to the cytosol.
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A novel, high conductance channel of mitochondria linked to apoptosis in mammalian cells and Bax expression in yeast.

Evgeny Pavlov, +8 more
- 26 Nov 2001 - 
TL;DR: Findings implicate this channel, named mitochondrial apoptosis–induced channel, as a candidate for the outer-membrane pore through which cytochrome c and possibly other factors exit mitochondria during apoptosis.