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Dave Bridges

Researcher at University of Michigan

Publications -  77
Citations -  11859

Dave Bridges is an academic researcher from University of Michigan. The author has contributed to research in topics: Insulin resistance & Internal medicine. The author has an hindex of 24, co-authored 68 publications receiving 10100 citations. Previous affiliations of Dave Bridges include Rambam Health Care Campus & University of Tennessee.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

14-3-3 proteins: a number of functions for a numbered protein.

Dave Bridges, +1 more
- 20 Jul 2004 - 
TL;DR: This STKE Review with 2 figures, 1 interactive molecular structure, and 118 references describes 14-3-3 proteins and highlights how these simple proteins have profound effects on the regulation of a vast number of cellular events.
Journal ArticleDOI

In vivo, Pikfyve generates PI(3,5)P2, which serves as both a signaling lipid and the major precursor for PI5P

Sergey N. Zolov, +13 more
- 23 Oct 2012 - 
TL;DR: Surprisingly, Pikfyve also is responsible for nearly all of the phosphatidylinositol-5-phosphate (PI5P) pool, and it is shown that PI5P is generated directly from PI(3,5)P2, likely via 3′-Phosphatase activity.
Journal ArticleDOI

Inhibition of AMPK catabolic action by GSK3

Tsukasa Suzuki, +7 more
- 09 May 2013 - 
TL;DR: It is reported that glycogen synthase kinase 3 (GSK3) inhibits AMPK function, revealing how AMPK senses anabolic environments in addition to cellular energy levels and acts as a critical sensor for anabolic signaling to regulate AMPK.