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Peter K. Kim

Researcher at University of Toronto

Publications -  93
Citations -  17003

Peter K. Kim is an academic researcher from University of Toronto. The author has contributed to research in topics: Peroxisome & Autophagy. The author has an hindex of 37, co-authored 77 publications receiving 13933 citations. Previous affiliations of Peter K. Kim include National Institutes of Health & Columbia University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Mitochondria Supply Membranes for Autophagosome Biogenesis during Starvation

TL;DR: It is demonstrated that, in starved cells, the outer membrane of mitochondria participates in autophagosome biogenesis, and Mitochondria play a central role in starvation-induced autophagy, contributing membrane to Autophagosomes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Ubiquitin signals autophagic degradation of cytosolic proteins and peroxisomes

TL;DR: In this article, simple mono-ubiquitination of normally long-lived cytoplasmic substrates is sufficient to target these substrates for autophagic degradation in mammalian cells.