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Marc Flajolet

Researcher at Rockefeller University

Publications -  76
Citations -  15534

Marc Flajolet is an academic researcher from Rockefeller University. The author has contributed to research in topics: Amyloid precursor protein & Receptor. The author has an hindex of 35, co-authored 70 publications receiving 13587 citations. Previous affiliations of Marc Flajolet include Scripps Research Institute.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Dichotomous Dopaminergic Control of Striatal Synaptic Plasticity

TL;DR: DA plays complementary roles in these two types of MSN to ensure that synaptic plasticity is bidirectional and Hebbian, leading to unidirectional changes in plasticity that could underlie network pathology and symptoms in Parkinson's disease.
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Pharmacological inhibitors of glycogen synthase kinase 3

TL;DR: A large body of evidence supports speculation that pharmacological inhibitors of GSK-3 could be used to treat several diseases, including Alzheimer's disease and other neurodegenerative diseases, bipolar affective disorder, diabetes, and diseases caused by unicellular parasites that express G SKS-3 homologues.
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Alterations in 5-HT1B Receptor Function by p11 in Depression-Like States

TL;DR: Overexpression of p11 increases 5-HT1B receptor function in cells and recapitulates certain behaviors seen after antidepressant treatment in mice, which contributes to depression-like phenotype and reduced behavioral reactions to an antidepressant.