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Maria Condello

Researcher at Istituto Superiore di Sanità

Publications -  48
Citations -  10775

Maria Condello is an academic researcher from Istituto Superiore di Sanità. The author has contributed to research in topics: Apoptosis & Autophagy. The author has an hindex of 19, co-authored 43 publications receiving 9232 citations. Previous affiliations of Maria Condello include Sapienza University of Rome & National Research Council.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Exposure to ZnO nanoparticles induces oxidative stress and cytotoxicity in human colon carcinoma cells.

Barbara De Berardis, +6 more
- 01 Aug 2010 - 
TL;DR: The experimental data show that oxidative stress may be a key route in inducing the cytotoxicity of ZnO nanoparticles in colon carcinoma cells, and the study of the relationship between toxicological effects and physico-chemical characteristics of particles suggests that surface area does not play a primary role in the cyttoxicity.
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Exosomes from human colorectal cancer induce a tumor-like behavior in colonic mesenchymal stromal cells.

Luana Lugini, +7 more
- 13 Jul 2016 - 
TL;DR: The results suggest that exosomes are actively involved in cancer progression and that inhibiting tumor exosome release may represent a way to interfere with cancer.
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The PPAR-γ agonist troglitazone antagonizes survival pathways induced by STAT-3 in recombinant interferon-β treated pancreatic cancer cells.

Giovanni Vitale, +16 more
- 01 Jan 2012 - 
TL;DR: To the best of the knowledge this is the first evidence that PPAR-γ activation can counteract STAT-3-dependent escape pathways to IFN-β-induced growth inhibition through cell cycle perturbation and increased autophagic death in pancreatic cancer cells.