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Cécile Vindis

Researcher at French Institute of Health and Medical Research

Publications -  60
Citations -  13696

Cécile Vindis is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Autophagy & Apoptosis. The author has an hindex of 37, co-authored 59 publications receiving 11165 citations. Previous affiliations of Cécile Vindis include University of Toulouse & Paul Sabatier University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Oxidized Low-Density Lipoproteins Trigger Endoplasmic Reticulum Stress in Vascular Cells Prevention by Oxygen-Regulated Protein 150 Expression

TL;DR: Investigation of whether ER stress is induced by oxLDLs and can be prevented by the ER-associated chaperone ORP150 found it to be prevented, as assessed by the protective effect of specific small interfering RNAs and c-Jun N-terminal kinase inhibitor.
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Ephrin-B1 transduces signals to activate integrin-mediated migration, attachment and angiogenesis

TL;DR: Ephrin-B1 transduces 'outside-in' signals through C-terminal protein interactions that affect integrin-mediated attachment and migration.