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Jin Wang

Researcher at Houston Methodist Hospital

Publications -  80
Citations -  14395

Jin Wang is an academic researcher from Houston Methodist Hospital. The author has contributed to research in topics: Apoptosis & Autophagy. The author has an hindex of 43, co-authored 78 publications receiving 12840 citations. Previous affiliations of Jin Wang include Children's Hospital Los Angeles & Baylor University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Mature T lymphocyte apoptosis--immune regulation in a dynamic and unpredictable antigenic environment.

Michael J. Lenardo, +6 more
- 01 Jan 1999 - 
TL;DR: Immunological, cellular, and molecular evidence indicates that throughout the life of a T cell, apoptosis may be evoked in excessive, harmful, or useless clonotypes to preserve a healthy and balanced immune system.
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Essential role for Nix in autophagic maturation of erythroid cells

Hector Sandoval, +6 more
- 10 Jul 2008 - 
TL;DR: It is suggested that Nix-dependent loss of ΔΨm is important for targeting the mitochondria into autophagosomes for clearance during erythroid maturation, and interference with this function impairs erythyroidmaturation and results in anaemia.
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Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency

Hyung J. Chun, +14 more
- 26 Sep 2002 - 
TL;DR: Caspase-8 deficiency in humans is compatible with normal development and shows that caspases has a postnatal role in immune activation of naive lymphocytes, which leads to immunodeficiency.
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Inherited Human Caspase 10 Mutations Underlie Defective Lymphocyte and Dendritic Cell Apoptosis in Autoimmune Lymphoproliferative Syndrome Type II

Jin Wang, +9 more
- 09 Jul 1999 - 
TL;DR: Results provide evidence that inherited nonlethal caspase abnormalities cause pleiotropic apoptosis defects underlying autoimmunity in ALPS type II.