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Youji Feng

Researcher at Shanghai Jiao Tong University

Publications -  54
Citations -  6190

Youji Feng is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Ovarian cancer & Endometrial cancer. The author has an hindex of 28, co-authored 51 publications receiving 5067 citations. Previous affiliations of Youji Feng include Fudan University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Snail is critical for tumor growth and metastasis of ovarian carcinoma

TL;DR: Gene array analysis revealed that blocking Snail expression suppressed the activity of matrix metalloproteinases (MMPs) and upregulated TIMP3, an MMP inhibitor, which suggests that Snail interacts with MMP during tumor invasion and metastasis.
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Inhibition of survivin reduces cell proliferation and induces apoptosis in human endometrial cancer.

TL;DR: Wang et al. as mentioned in this paper used RNA interference to knock down survivin expression in the Ishikawa endometrial cancer cell line by recombinant plasmids producing survivin small hairpin RNA.
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Estrogen induces endometrial cancer cell proliferation and invasion by regulating the fat mass and obesity-associated gene via PI3K/AKT and MAPK signaling pathways.

TL;DR: In this article, expression of the fat mass and obesity-associated (FTO) gene in endometrial tumor tissues and further explored its role in β-estradiol (E2)-induced endometrium cancer cell proliferation and invasion.
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Nuclear estrogen receptor-mediated Notch signaling and GPR30-mediated PI3K/AKT signaling in the regulation of endometrial cancer cell proliferation.

TL;DR: Conclusively, regulation of proliferation in nuclear ER-positive endometrial cancer cells is mediated by both ER-Notch signaling and GPR30-PI3K/AKT signaling, whereas only the latter pathway is involved in the regulation of growth in nuclear EMTs.