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Thirumala-Devi Kanneganti

Researcher at St. Jude Children's Research Hospital

Publications -  339
Citations -  50046

Thirumala-Devi Kanneganti is an academic researcher from St. Jude Children's Research Hospital. The author has contributed to research in topics: Inflammasome & Innate immune system. The author has an hindex of 94, co-authored 305 publications receiving 37831 citations. Previous affiliations of Thirumala-Devi Kanneganti include Ohio Agricultural Research and Development Center & Boston Children's Hospital.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Cytosolic flagellin requires Ipaf for activation of caspase-1 and interleukin 1β in salmonella-infected macrophages

TL;DR: The authors showed that salmonella-infected and lipopolysaccharide-tolerant macrophages were deficient in activation of caspase-1 and in interleukin 1beta secretion, although transcription factor NF-kappaB-dependent production of the chemokine MCP-1 was unimpaired.