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Jean X. Jiang

Researcher at University of Texas Health Science Center at San Antonio

Publications -  160
Citations -  13212

Jean X. Jiang is an academic researcher from University of Texas Health Science Center at San Antonio. The author has contributed to research in topics: Connexin & Gap junction. The author has an hindex of 47, co-authored 160 publications receiving 10865 citations. Previous affiliations of Jean X. Jiang include University of Texas System & University of Texas at Austin.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Mechanical strain opens connexin 43 hemichannels in osteocytes: a novel mechanism for the release of prostaglandin.

Priscilla P. Cherian, +6 more
- 01 Jul 2005 - 
TL;DR: Results suggest fluid flow shear stress induces the translocation of Cx43 to the membrane surface and that unapposed hemichannels formed by Cx 43 serve as a novel portal for the release of PGE2 in response to mechanical strain.
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Negative feedback control of neuronal activity by microglia

Ana Badimon, +32 more
- 15 Oct 2020 - 
TL;DR: It is suggested that this microglia-driven negative feedback mechanism operates similarly to inhibitory neurons and is essential for protecting the brain from excessive activation in health and disease.
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ATP and glutamate released via astroglial connexin 43 hemichannels mediate neuronal death through activation of pannexin 1 hemichannels

Juan A. Orellana, +9 more
- 07 Mar 2011 - 
TL;DR: In this article, the role of microglia treated with amyloid-β peptide (Aβ) on hemichannel activity of astrocytes subjected to hypoxia in high glucose was studied.
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Reactive oxygen species and oxidative stress in osteoclastogenesis, skeletal aging and bone diseases

Danielle A. Callaway, +1 more
- 26 Mar 2015 - 
TL;DR: The mechanisms by which ROS can be produced in osteoclasts as well as how it may affect osteoclast differentiation and activity will be examined and how targeting ROS may lead to the development of novel therapeutic treatment options.