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Bärbel Rohrer

Researcher at Medical University of South Carolina

Publications -  97
Citations -  14566

Bärbel Rohrer is an academic researcher from Medical University of South Carolina. The author has contributed to research in topics: Complement system & Alternative complement pathway. The author has an hindex of 32, co-authored 93 publications receiving 12761 citations. Previous affiliations of Bärbel Rohrer include Veterans Health Administration.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Daniel J. Klionsky, +235 more
- 16 Feb 2008 - 
TL;DR: A set of guidelines for the selection and interpretation of the methods that can be used by investigators who are attempting to examine macroautophagy and related processes, as well as by reviewers who need to provide realistic and reasonable critiques of papers that investigate these processes are presented.
Journal ArticleDOI

Oxidative stress renders retinal pigment epithelial cells susceptible to complement-mediated injury.

TL;DR: It is demonstrated that oxidative stress reduces regulation of complement on the surface of ARPE-19 cells, increasing complement activation and linking oxidative stress, complement activation, and apical VEGF release, which have all been associated with the pathogenesis of AMD.
Journal ArticleDOI

Apoptosis and autophagy in photoreceptors exposed to oxidative stress.

TL;DR: It is confirmed that cells that normally die by apoptosis will execute cell death by necrosis if the normal pathway is blocked, and the up-stream regulators of autophagy need to be identified as potential therapeutic targets in photoreceptor degeneration.