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Ricardo Sánchez-Prieto

Researcher at University of Castilla–La Mancha

Publications -  78
Citations -  11972

Ricardo Sánchez-Prieto is an academic researcher from University of Castilla–La Mancha. The author has contributed to research in topics: Signal transduction & Cancer. The author has an hindex of 25, co-authored 70 publications receiving 10415 citations. Previous affiliations of Ricardo Sánchez-Prieto include Carlos III Health Institute & National Institutes of Health.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal Article

A Role for the p38 Mitogen-activated Protein Kinase Pathway in the Transcriptional Activation of p53 on Genotoxic Stress by Chemotherapeutic Agents

Ricardo Sánchez-Prieto, +3 more
- 01 May 2000 - 
TL;DR: It is found that blockade of p38 prevents stimulation of the transcriptional activity of p53 and that activation of the p38 pathway is sufficient to stimulate p53 function, suggesting a role for p38 activation in the apoptotic response to genotoxic stress when elicited by drugs used in cancer therapy.
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Full activation of PKB/Akt in response to insulin or ionizing radiation is mediated through ATM.

Juan Guinea Viniegra, +10 more
- 11 Feb 2005 - 
TL;DR: It is shown that ATM is a major determinant of full PKB/Akt activation in response to insulin or γ-radiation, and biological responses tightly regulated by Akt were altered in cell lines derived from AT patients and knockout mice for ATM.
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HIF2α Acts as an mTORC1 Activator through the Amino Acid Carrier SLC7A5

Ainara Elorza, +18 more
- 14 Dec 2012 - 
TL;DR: A link between the oxygen-sensing HIF2α pathway and mTORC1 regulation is identified, revealing the molecular basis of the tumor-promoting properties of Hif2α in von Hippel-Lindau-deficient cells.