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Lorrie A. Kirshenbaum

Researcher at University of Manitoba

Publications -  164
Citations -  19290

Lorrie A. Kirshenbaum is an academic researcher from University of Manitoba. The author has contributed to research in topics: Programmed cell death & Apoptosis. The author has an hindex of 51, co-authored 151 publications receiving 16234 citations. Previous affiliations of Lorrie A. Kirshenbaum include University of Western Ontario & St. Boniface General Hospital.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy.

TL;DR: The results suggest that Bnip3 contributes to I/R injury which triggers a protective stress response with upregulation of autophagy and removal of damaged mitochondria.
Journal ArticleDOI

Multiple Facets of NF-κB in the Heart To Be or Not to NF-κB

TL;DR: A novel paradigm is posit in which the timing, duration of activation, and cellular context may explain mechanistically the differential outcomes of NF-κB signaling in the heart that may be essential for future development of novel therapeutic interventions designed to target NF-σB responses and heart failure following myocardial injury.