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Clarissa von Haefen

Researcher at Charité

Publications -  59
Citations -  12244

Clarissa von Haefen is an academic researcher from Charité. The author has contributed to research in topics: Apoptosis & Oxidative stress. The author has an hindex of 22, co-authored 51 publications receiving 9905 citations. Previous affiliations of Clarissa von Haefen include Humboldt University of Berlin & Free University of Berlin.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Ceramide induces mitochondrial activation and apoptosis via a Bax-dependent pathway in human carcinoma cells

Clarissa von Haefen, +6 more
- 06 Jun 2002 - 
TL;DR: It is shown by measurement of mitochondrial permeability transition, cytochrome c release, activation of caspase-3 and DNA fragmentation that ceramide-induced apoptosis is marginal in Bax-negative DU 145 cells, and it is concluded that Bax is a key activator of Ceramide-mediated death pathways.
Journal ArticleDOI

Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop

Clarissa von Haefen, +5 more
- 17 Apr 2003 - 
TL;DR: It is shown that caspases-3 and -8 mediate a mitochondrial amplification loop that is required for the optimal release of cytochrome c, mitochondrial permeability shift transition, and cell death during apoptosis induced by treatment with the microtubule-damaging agent paclitaxel (Taxol).