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Behzad Yeganeh

Researcher at University of Toronto

Publications -  49
Citations -  13046

Behzad Yeganeh is an academic researcher from University of Toronto. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 28, co-authored 47 publications receiving 11228 citations. Previous affiliations of Behzad Yeganeh include University of Manitoba & Boston Children's Hospital.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Autophagy and apoptosis dysfunction in neurodegenerative disorders.

Saeid Ghavami, +12 more
- 01 Jan 2014 - 
TL;DR: In this article, the authors provide a brief introduction to autophagy and apoptosis pathways focusing on the role of mitochondria and lysosomes, and discuss different ways that autophag and apoptotic modulation may be employed for therapeutic intervention during the maintenance of neurodegenerative disorders.
Journal ArticleDOI

Apoptosis and cancer: mutations within caspase genes

Saeid Ghavami, +10 more
- 01 Aug 2009 - 
TL;DR: This review gives an overview of the role of caspases, their natural modulators like IAPs, FLIPs, and Smac/Diablo in apoptosis and upon inactivation, and also in cancer development and some of the direct activators of procaspase-3.
Journal ArticleDOI

Autophagy is a regulator of TGF-β1-induced fibrogenesis in primary human atrial myofibroblasts.

Saeid Ghavami, +11 more
- 19 Mar 2015 - 
TL;DR: The hypothesis that TGF-β1-induced autophagy is required for the fibrogenic response in hATMyofbs is supported.