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Antonio Musarò

Researcher at Sapienza University of Rome

Publications -  179
Citations -  15117

Antonio Musarò is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Skeletal muscle & Muscle atrophy. The author has an hindex of 46, co-authored 169 publications receiving 12976 citations. Previous affiliations of Antonio Musarò include Victor Chang Cardiac Research Institute & Pierre-and-Marie-Curie University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Localized Igf-1 transgene expression sustains hypertrophy and regeneration in senescent skeletal muscle

Antonio Musarò, +8 more
- 01 Feb 2001 - 
TL;DR: A model of persistent, functional myocyte hypertrophy is generated using a tissue-restricted transgene encoding a locally acting isoform of insulin-like growth factor-1 that is expressed in skeletal muscle (mIgf-1) and suggests clinical strategies for the treatment of age or disease-related muscle frailty.
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Viral mediated expression of insulin-like growth factor I blocks the aging-related loss of skeletal muscle function

Elisabeth R. Barton-Davis, +4 more
- 22 Dec 1998 - 
TL;DR: The results suggest that gene transfer of IGF-I into muscle could form the basis of a human gene therapy for preventing the loss of muscle function associated with aging and may be of benefit in diseases where the rate of damage to skeletal muscle is accelerated.
Journal ArticleDOI

IGF-1 induces skeletal myocyte hypertrophy through calcineurin in association with GATA-2 and NF-ATc1

Antonio Musarò, +4 more
- 05 Aug 1999 - 
TL;DR: It is shown that the molecular pathways underlying the hypertrophic action of IGF-1 in skeletal muscle are similar to those responsible for cardiac hypertrophy.
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Muscle-specific expression of insulin-like growth factor I counters muscle decline in mdx mice.

Elisabeth R. Barton, +4 more
- 01 Apr 2002 - 
TL;DR: Results suggest that a combination of promoting muscle regenerative capacity and preventing muscle necrosis could be an effective treatment for the secondary symptoms caused by the primary loss of dystrophin.