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Robert Gilkerson

Researcher at The University of Texas Rio Grande Valley

Publications -  33
Citations -  5530

Robert Gilkerson is an academic researcher from The University of Texas Rio Grande Valley. The author has contributed to research in topics: Mitochondrial DNA & Mitochondrion. The author has an hindex of 14, co-authored 29 publications receiving 4376 citations. Previous affiliations of Robert Gilkerson include Columbia University & University of Texas–Pan American.

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Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Development of tannic acid/chitosan/pullulan composite nanofibers from aqueous solution for potential applications as wound dressing.

Fenghua Xu, +4 more
- 22 Jan 2015 - 
TL;DR: This novel membrane favors fibroblast cell attachment and growth by providing a 3D environment which mimics the extracellular matrix (ECM) in skin and allows cells to move through the fibrous structure resulting in interlayer growth throughout the membrane, thus favoring potential for deep and intricate wound healing.
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Past, Present and Future of Surgical Meshes: A Review.

Karen Baylón, +5 more
TL;DR: An overview of commercial surgical meshes, their properties, manufacturing methods, and observed biological response are presented, as well as the requirements for an ideal surgical mesh and potential manufacturing methods are presented.
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Mitochondrial nucleoids maintain genetic autonomy but allow for functional complementation.

Robert Gilkerson, +3 more
- 30 Jun 2008 - 
TL;DR: Results indicate that mitochondrial nucleoids tightly regulate their genetic content rather than freely exchanging mtDNAs, which provides a molecular mechanism to explain patterns of mitochondrial genetic inheritance, in addition to facilitating therapeutic methods to eliminate deleterious mtDNA mutations.
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Ketogenic treatment reduces deleted mitochondrial DNAs in cultured human cells

Sumana Santra, +3 more
- 01 Nov 2004 - 
TL;DR: The demonstration that ketone bodies can distinguish between normal and respiratorily compromised cells points to the potential use of a ketogenic diet to treat patients with heteroplasmic mtDNA disorders.