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Lixin Wei

Researcher at Second Military Medical University

Publications -  145
Citations -  9993

Lixin Wei is an academic researcher from Second Military Medical University. The author has contributed to research in topics: Autophagy & Apoptosis. The author has an hindex of 38, co-authored 134 publications receiving 8079 citations. Previous affiliations of Lixin Wei include Shanghai Jiao Tong University & University of Medicine and Dentistry of New Jersey.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Tumor-associated macrophages promote cancer stem cell-like properties via transforming growth factor-beta1-induced epithelial-mesenchymal transition in hepatocellular carcinoma.

Qing-Min Fan, +10 more
- 01 Oct 2014 - 
TL;DR: It is found that the number of TAMs has a positive correlation with the density of CSCs in the marginal of human HCC and they may contribute to investigate the prognosis of HCC.
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Epithelial-Mesenchymal Transition in tumor microenvironment

Yingying Jing, +4 more
- 31 Aug 2011 - 
TL;DR: The tumor microenvironment plays an important role in facilitating cancer metastasis and may induce the occurrence of EMT in tumor cells, including TGF-β, NF-κB, Wnt, Notch, and others.
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Hypoxia-induced autophagy contributes to the chemoresistance of hepatocellular carcinoma cells.

Jianrui Song, +9 more
- 16 Nov 2009 - 
TL;DR: Autophagy decreases hepatoma cells sensitization to chemotherapeutic agents by affecting their apoptotic potential, which means autophagy mediates the chemoresistance under hypoxia.
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Effects of inflammatory factors on mesenchymal stem cells and their role in the promotion of tumor angiogenesis in colon cancer.

Yan Liu, +13 more
- 15 Jul 2011 - 
TL;DR: Results indicate that MSCs stimulated by inflammatory cytokines such as IFN-γ and TNF-α in the tumor microenvironment express higher levels of VEGF via the HIF-1α signaling pathway and that these MSCS then enhance tumor angiogenesis, finally leading to colon cancer growth in mice.