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Henri Bernardi

Researcher at University of Montpellier

Publications -  37
Citations -  6453

Henri Bernardi is an academic researcher from University of Montpellier. The author has contributed to research in topics: Skeletal muscle & Autophagy. The author has an hindex of 20, co-authored 36 publications receiving 5393 citations. Previous affiliations of Henri Bernardi include Centre national de la recherche scientifique & Institut national de la recherche agronomique.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Mechanisms involved in the inhibition of myoblast proliferation and differentiation by myostatin.

TL;DR: The data show the influence of overexpression on myoblast proliferation, differentiation, and apoptosis is extended to endogenous myostatin, and it appears that myogenin and p21 are essential targets of this growth factor.
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AMPK promotes skeletal muscle autophagy through activation of forkhead FoxO3a and interaction with Ulk1.

TL;DR: The data show that AMPK activation stimulates autophagy in skeletal muscle cells through its effects on the transcriptional function of FoxO3a and takes part in the initiation of autophagosome formation by interacting with Ulk1.
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Antidiabetic sulfonylureas: localization of binding sites in the brain and effects on the hyperpolarization induced by anoxia in hippocampal slices.

TL;DR: Electrophysiological studies with hippocampal slices show that glibenclamide blocks hyperpolarization induced by anoxia, suggesting the involvement of adenosine triphosphate-sensitive K+ channel in this early hyperpolarsization event.
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FoxO transcription factors: their roles in the maintenance of skeletal muscle homeostasis

TL;DR: The multiple modes of FoxO regulation are detailed and the central role of these proteins in the control of skeletal muscle plasticity is discussed, as well as the role these proteins have in the regulation of the cell cycle, apoptosis, and muscle regeneration.