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Byung Wan Lee

Researcher at Yonsei University

Publications -  277
Citations -  10848

Byung Wan Lee is an academic researcher from Yonsei University. The author has contributed to research in topics: Diabetes mellitus & Type 2 diabetes. The author has an hindex of 36, co-authored 255 publications receiving 8167 citations. Previous affiliations of Byung Wan Lee include Severance Hospital & Hallym University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Background and Data Configuration Process of a Nationwide Population-Based Study Using the Korean National Health Insurance System

TL;DR: The history, structure, contents, and way to use data procurement in the Korean National Health Insurance (NHI) system for the benefit of Korean researchers makes it a world-leading population-based epidemiology and disease research platform.
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Sarcopenia is associated with significant liver fibrosis independently of obesity and insulin resistance in nonalcoholic fatty liver disease: Nationwide surveys (KNHANES 2008-2011)

TL;DR: Sarcopenia is associated with significant liver fibrosis in subjects with NAFLD, and the association is independent of obesity and insulin resistance.
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Sarcopaenia is associated with NAFLD independently of obesity and insulin resistance: Nationwide surveys (KNHANES 2008-2011)

TL;DR: Sarcopaenia is associated with increased risks of non-alcoholic fatty liver disease (NAFLD) and advanced fibrosis, independent of obesity or metabolic control.
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Metformin alleviates hepatosteatosis by restoring SIRT1-mediated autophagy induction via an AMP-activated protein kinase-independent pathway

TL;DR: Results suggest that metformin alleviates hepatic steatosis through PRKA-independent, SIRT1-mediated effects on the autophagy machinery.