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Jae-Young Koh

Researcher at University of Ulsan

Publications -  186
Citations -  24214

Jae-Young Koh is an academic researcher from University of Ulsan. The author has contributed to research in topics: Neurotoxicity & Kainate receptor. The author has an hindex of 65, co-authored 184 publications receiving 22144 citations. Previous affiliations of Jae-Young Koh include Asan Medical Center & Korea Institute of Science and Technology.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The neurobiology of zinc in health and disease

Christopher J. Frederickson, +3 more
- 01 Jun 2005 - 
TL;DR: The use of zinc in medicinal skin cream was mentioned in Egyptian papyri from 2000 BC, and the number of biological functions, health implications and pharmacological targets that are emerging for zinc indicate that it might turn out to be 'the calcium of the twenty-first century'.
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Quantitative determination of glutamate mediated cortical neuronal injury in cell culture by lactate dehydrogenase efflux assay

Jae-Young Koh, +1 more
- 01 May 1987 - 
TL;DR: In this paper, a simple yet quantitative method for assessing glutamate mediated central neuronal cell injury in cortical cell culture has been proposed; the magnitude of LDH efflux in the cultures correlates in a linear fashion with the number of neurons damaged by glutamate exposure.
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The Role of Zinc in Selective Neuronal Death After Transient Global Cerebral Ischemia

Jae-Young Koh, +5 more
- 17 May 1996 - 
TL;DR: The toxic influx of zinc may be a key mechanism underlying selective neuronal death after transient global ischemic insults and could be prevented by the intraventricular injection of a zinc chelating agent.
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Pharmacology of glutamate neurotoxicity in cortical cell culture: attenuation by NMDA antagonists

Dennis W. Choi, +2 more
- 01 Jan 1988 - 
TL;DR: The present study suggests that glutamate neurotoxicity may be predominantly mediated by the activation of the NMDA subclass of glutamate receptors--occurring both directly, during exposure to exogenous compound, and indirectly, due to the subsequent release of endogenous NMDA agonists.