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Nadia Zaffaroni

Researcher at University of Milan

Publications -  369
Citations -  24282

Nadia Zaffaroni is an academic researcher from University of Milan. The author has contributed to research in topics: Cancer & Apoptosis. The author has an hindex of 58, co-authored 342 publications receiving 21661 citations. Previous affiliations of Nadia Zaffaroni include European Organisation for Research and Treatment of Cancer & National Institutes of Health.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Isolation and in vitro propagation of tumorigenic breast cancer cells with stem/progenitor cell properties.

TL;DR: Long-term cultures of breast tumorigenic cells with stem/progenitor cell properties represent a suitable in vitro model to study breast cancer-initiating cells and to develop therapeutic strategies aimed at eradicating the tumorigenics subpopulation within breast cancer.
Journal ArticleDOI

miR-205 Exerts Tumor-Suppressive Functions in Human Prostate through Down-regulation of Protein Kinase Cε

TL;DR: Overall, it is shown for the first time that miR-205 exerts a tumor-suppressive effect in human prostate by counteracting epithelial-to-mesenchymal transition and reducing cell migration/invasion, at least in part through the down-regulation of protein kinase Cepsilon.