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Uma Sankar

Researcher at Indiana University

Publications -  36
Citations -  5448

Uma Sankar is an academic researcher from Indiana University. The author has contributed to research in topics: Cellular differentiation & Chemistry. The author has an hindex of 14, co-authored 27 publications receiving 4415 citations. Previous affiliations of Uma Sankar include Ohio State University & Duke University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Microphthalmia transcription factor is a target of the p38 MAPK pathway in response to receptor activator of NF-κB ligand signaling

Kim C. Mansky, +3 more
- 29 Mar 2002 - 
TL;DR: Results indicate that MITF is a target for the RANKL signaling pathway in osteoclasts and that phosphorylation of MITF leads to an increase in osteoclast-specific gene expression.
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Identification of Adiponectin as a Novel Hemopoietic Stem Cell Growth Factor

Leah N. DiMascio, +7 more
- 15 Mar 2007 - 
TL;DR: Investigation of the role of adiponectin in hemopoietic stem cell function identifies it as a novel regulator of HSC function and suggests that it acts through a p38 dependent pathway.
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A novel Gfer-Drp1 link in preserving mitochondrial dynamics and function in pluripotent stem cells.

Lance R. Todd, +5 more
- 01 Apr 2010 - 
TL;DR: Gfer links mitochondrial dynamics with pluripotency in mouse embryonic stem cells through its modulation of the mitochondrial fission GTPase Drp1.
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Eos, MITF, and PU.1 recruit corepressors to osteoclast-specific genes in committed myeloid progenitors.

Rong Hu, +5 more
- 01 Jun 2007 - 
TL;DR: A mechanism to account for the modulation of MITF and PU.1 activity in committed myeloid progenitors prior to the initiation of osteoclast differentiation in response to the appropriate extracellular signals is provided.