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Francesca Rovetta

Researcher at University of Brescia

Publications -  11
Citations -  4708

Francesca Rovetta is an academic researcher from University of Brescia. The author has contributed to research in topics: Autophagy & Skeletal muscle. The author has an hindex of 9, co-authored 11 publications receiving 3783 citations. Previous affiliations of Francesca Rovetta include Brescia University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Modulation of redox status and calcium handling by extremely low frequency electromagnetic fields in C2C12 muscle cells: A real-time, single-cell approach

TL;DR: The data support a possible link between exposure to ELF-EMFs and modification of the cellular redox state, which could, in turn, increase the level of intracellular Ca(2+) and thus modulate the metabolic activity of C2C12 cells.
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In vivo heat-shock response in the brain: signalling pathway and transcription factor activation.

TL;DR: The data indicate that a physiologically relevant increase in body temperature induces brain injury and survival response to it as demonstrated by induction of hsp70 gene expression and activation of specific signalling pathways.
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Cisplatin triggers atrophy of skeletal C2C12 myotubes via impairment of Akt signalling pathway and subsequent increment activity of proteasome and autophagy systems.

TL;DR: Results indicate that cisPt induces atrophy of C2C12 myotubes via activation of proteasome and autophagy systems, suggesting that the Akt pathway represents one sensitive target of cisPT molecular action in skeletal muscle.
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Endogenous thiols and MRP transporters contribute to Hg2+ efflux in HgCl2-treated tubular MDCK cells.

TL;DR: The results demonstrate that, in MDCK cells, inorganic Hg(2+) promotes the activation of specific detoxifying pathways that may, at least partly, depend on the activity of MRP transporters.