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Hiroyasu Nakano

Researcher at Toho University

Publications -  154
Citations -  22736

Hiroyasu Nakano is an academic researcher from Toho University. The author has contributed to research in topics: Programmed cell death & Signal transduction. The author has an hindex of 57, co-authored 143 publications receiving 20363 citations. Previous affiliations of Hiroyasu Nakano include Chiba University & Juntendo University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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ASK1 Is Essential for JNK/SAPK Activation by TRAF2

Hideki Nishitoh, +11 more
- 01 Sep 1998 - 
TL;DR: It is shown that ASK1 interacts with members of the TRAF family and is activated by TRAF2, TRAF5, and TRAF6 overexpression and is a mediator of TRAf2-induced JNK activation.
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SHARPIN is a component of the NF-κB-activating linear ubiquitin chain assembly complex

Fuminori Tokunaga, +8 more
- 31 Mar 2011 - 
TL;DR: Deletion of SHARPIN drastically reduced the amount of LUBAC, which resulted in attenuated TNF-α- and CD40-mediated activation of NF-κB in mouse embryonic fibroblasts (MEFs) or B cells from cpdm mice, and strongly suggest the involvement of LubAC-induced NF-σκB activation in various disorders.
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Differential regulation of IκB kinase α and β by two upstream kinases, NF-κB-inducing kinase and mitogen-activated protein kinase/ERK kinase kinase-1

Hiroyasu Nakano, +6 more
- 31 Mar 1998 - 
TL;DR: Results indicate that Nik and MEKK1 independently activate the IKK complex and that the kinase activities of IKKalpha and IKKbeta are differentially regulated by two upstream kinases, NIK andMEKK1, which are responsive to distinct stimuli.