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Marco Tafani

Researcher at Sapienza University of Rome

Publications -  96
Citations -  15553

Marco Tafani is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Apoptosis & Programmed cell death. The author has an hindex of 35, co-authored 96 publications receiving 12797 citations. Previous affiliations of Marco Tafani include University of Rome Tor Vergata & Thomas Jefferson University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

The Overexpression of Bax Produces Cell Death upon Induction of the Mitochondrial Permeability Transition

John G. Pastorino, +4 more
- 27 Mar 1998 - 
TL;DR: It is concluded that Bax induces the mitochondrial permeability transition (MPT), a critical event in the loss of cell viability, and mediates other typical manifestations of apoptosis in this model, namely release of cytochrome c, caspase activation with PARP cleavage, and DNA fragmentation.
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The course of etoposide-induced apoptosis from damage to DNA and p53 activation to mitochondrial release of cytochrome c.

Natalie O. Karpinich, +5 more
- 10 May 2002 - 
TL;DR: A sequence of biochemical events that mediates the apoptosis induced by etoposide is defined, which proceeds by coupling DNA damage to p53 phosphorylation through the action of DNA-PK and releases cytochrome c and culminates in the death of the cells.