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Linda Sealy

Researcher at Vanderbilt University

Publications -  55
Citations -  7953

Linda Sealy is an academic researcher from Vanderbilt University. The author has contributed to research in topics: Transcription factor & Ccaat-enhancer-binding proteins. The author has an hindex of 31, co-authored 55 publications receiving 6958 citations. Previous affiliations of Linda Sealy include University of Iowa & University of California, San Francisco.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The effect of sodium butyrate on histone modification

Linda Sealy, +1 more
- 01 May 1978 - 
TL;DR: The hyperacetylation of histones due to treatment of cultured cells with sodium butyrate has been studied and it is found that 80% of histone H4 is acetylated after a 24 hr exposure tobutyrate.
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A novel role for p120 catenin in E-cadherin function.

Reneé C. Ireton, +12 more
- 11 Nov 2002 - 
TL;DR: The p120 gene is mutated in SW48 cells, and that the cadherin adhesion system is impaired as a direct consequence of p120 insufficiency, indicating a crucial role for p120 in reactivation of E-cadherin function.
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Bile Acid Regulation of C/EBPβ, CREB, and c-Jun Function, via the Extracellular Signal-Regulated Kinase and c-Jun NH2-Terminal Kinase Pathways, Modulates the Apoptotic Response of Hepatocytes

Liang Qiao, +12 more
- 01 May 2003 - 
TL;DR: Analysis of the roles of the ERK and c-Jun NH2-terminal kinase (JNK) pathways, and downstream transcription factors, in the survival response of hepatocytes demonstrated that multiple DCA-induced signaling pathways and transcription factors control hepatocyte survival.
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The alternatively initiated c-Myc proteins differentially regulate transcription through a noncanonical DNA-binding site.

Stephen R. Hann, +3 more
- 15 Oct 1994 - 
TL;DR: New transcriptional activities for the Myc proteins are revealed and it is demonstrated that the different forms of the MyC protein are functionally distinct, suggesting an interplay between two different growth regulatory transcription factor families.