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Hyung Ryong Kim

Researcher at Daegu Gyeongbuk Institute of Science and Technology

Publications -  33
Citations -  11075

Hyung Ryong Kim is an academic researcher from Daegu Gyeongbuk Institute of Science and Technology. The author has contributed to research in topics: Unfolded protein response & Endoplasmic reticulum. The author has an hindex of 17, co-authored 30 publications receiving 9495 citations. Previous affiliations of Hyung Ryong Kim include Wonkwang University & Sanford-Burnham Institute for Medical Research.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

ER stress: Autophagy induction, inhibition and selection.

Harun Or Rashid, +3 more
- 21 Sep 2015 - 
TL;DR: The crosstalk between ER stress and autophagy and their signaling networks mainly in mammalian-based systems is summarized and current knowledge on selective autophagic and its connection to ER stress is highlighted.
Journal ArticleDOI

BI-1 Regulates an Apoptosis Pathway Linked to Endoplasmic Reticulum Stress

Han-Jung Chae, +17 more
- 13 Aug 2004 - 
TL;DR: In vivo, bi-1(-/-) mice exhibit increased sensitivity to tissue damage induced by stimuli that trigger ER stress, including stroke and tunicamycin injection, and BI-1 regulates a cell death pathway important for cytopreservation during ER stress.
Journal ArticleDOI

Bax Inhibitor-1 Is a pH-dependent Regulator of Ca2+ Channel Activity in the Endoplasmic Reticulum

Hyung Ryong Kim, +12 more
- 06 Jun 2008 - 
TL;DR: It is suggested that Bax inhibitor-1 increases Ca2+ leak rates from the ER through a mechanism that is dependent on pH and on the carboxyl-terminal cytosolic region of the BI-1 protein.