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László Fésüs

Researcher at University of Debrecen

Publications -  277
Citations -  22572

László Fésüs is an academic researcher from University of Debrecen. The author has contributed to research in topics: Tissue transglutaminase & Apoptosis. The author has an hindex of 52, co-authored 268 publications receiving 20623 citations. Previous affiliations of László Fésüs include University of Texas Health Science Center at Houston & French Institute of Health and Medical Research.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Daniel J. Klionsky, +235 more
- 16 Feb 2008 - 
TL;DR: A set of guidelines for the selection and interpretation of the methods that can be used by investigators who are attempting to examine macroautophagy and related processes, as well as by reviewers who need to provide realistic and reasonable critiques of papers that investigate these processes are presented.
Journal ArticleDOI

Transglutaminase 2: an enigmatic enzyme with diverse functions

László Fésüs, +1 more
- 01 Oct 2002 - 
TL;DR: An important role for the enzyme in the pathogenesis of coeliac disease, fibrosis and neurodegenerative disorders has also been demonstrated, making TG2 an important therapeutic target.
Journal ArticleDOI

Induction and activation of tissue transglutaminase during programmed cell death

László Fésüs, +2 more
- 16 Nov 1987 - 
TL;DR: During the involution of lead nitrate‐induced hyperplasia in rat liver a significant increase of transglutaminase activity, enzyme concentration, transglUTaminase messenger RNA and protein‐bound ε‐(γ‐glutamyl)lysine (product of transGlutaminases action) coincided with programmed death (apoptosis) of hepatocytes.