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Beat Bornhauser

Researcher at Boston Children's Hospital

Publications -  94
Citations -  8863

Beat Bornhauser is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Leukemia & Medicine. The author has an hindex of 27, co-authored 81 publications receiving 6375 citations. Previous affiliations of Beat Bornhauser include Uppsala University & Hannover Medical School.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Down syndrome acute lymphoblastic leukemia, a highly heterogeneous disease in which aberrant expression of CRLF2 is associated with mutated JAK2: a report from the International BFM Study Group

Libi Hertzberg, +31 more
- 04 Feb 2010 - 
TL;DR: The data suggest that the majority of DS children with ALL may benefit from therapy blocking the CRLF2/JAK2 pathways, and the gene expression signature of DS-ALL is enriched with DNA damage and BCL6 responsive genes, suggesting the possibility of B-cell lymphocytic genomic instability.
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Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance

Laura Bonapace, +9 more
- 01 Apr 2010 - 
TL;DR: Complete resensitization of multidrug-resistant childhood ALL cells to glucocorticoids and other cytotoxic agents with subcytotoxic concentrations of obatoclax, a putative antagonist of BCL-2 family members is reported.
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Short communication: altered synaptic clustering of GABAA receptors in mice lacking dystrophin (mdx mice).

Irene Knuesel, +5 more
- 01 Dec 1999 - 
TL;DR: This article showed that dystrophin is extensively colocalized with GABAA receptor subunit clusters in cerebral cortex, hippocampus and cerebellum of mice with Duchenne muscular dystrophy.