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Ho Jeong Kwon

Researcher at Yonsei University

Publications -  226
Citations -  14196

Ho Jeong Kwon is an academic researcher from Yonsei University. The author has contributed to research in topics: Angiogenesis & Tube formation. The author has an hindex of 45, co-authored 219 publications receiving 11608 citations. Previous affiliations of Ho Jeong Kwon include Sejong University & National Institutes of Health.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Histone deacetylases induce angiogenesis by negative regulation of tumor suppressor genes.

TL;DR: It is indicated that hypoxia enhances HDAC function and that HDAC is closely involved in angiogenesis through suppression of Hypoxia-responsive tumor suppressor genes.
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Induction of cell cycle arrest and apoptosis in human breast cancer cells by quercetin.

TL;DR: It is demonstrated that a flavonoid quercetin induces growth inhibition in the human breast carcinoma cell line MCF-7 through at least two different mechanisms; by inhibiting cell cycle progression through transient M phase accumulation and subsequent G2 arrest, and by inducing apoptosis.
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Expression profile of histone deacetylase 1 in gastric cancer tissues.

TL;DR: It is clearly demonstrates that HDAC1 is overexpressed in GC and probably plays a significant role in gastric carcinogenesis.