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Feng Han

Researcher at Tohoku University

Publications -  34
Citations -  5800

Feng Han is an academic researcher from Tohoku University. The author has contributed to research in topics: Ca2+/calmodulin-dependent protein kinase & Cholinergic neuron. The author has an hindex of 21, co-authored 33 publications receiving 4794 citations. Previous affiliations of Feng Han include Michigan State University & Zhejiang University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Neuronal injury in rat model of permanent focal cerebral ischemia is associated with activation of autophagic and lysosomal pathways

Ya-Dan Wen, +8 more
- 20 Jun 2008 - 
TL;DR: The results demonstrate that ischemic insult activates autophagy and an autophagic mechanism may contribute to isChemic neuronal injury and may be a potential target for developing a novel therapy for stroke.
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p53 mediates mitochondria dysfunction-triggered autophagy activation and cell death in rat striatum.

Xingding Zhang, +10 more
- 01 Apr 2009 - 
TL;DR: The results suggest that p53 plays roles in signaling both autophagy and apoptosis, at least partially, contributes to neurodegeneration induced by mitochondria dysfunction.
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Constitutively active calcineurin mediates delayed neuronal death through Fas-ligand expression via activation of NFAT and FKHR transcriptional activities in mouse brain ischemia.

Norifumi Shioda, +3 more
- 01 Sep 2007 - 
TL;DR: The results suggest that FK HR is a downstream target of CaN and that constitutively active CaN mediates delayed neuronal death through Fas‐ligand expression via up‐regulation of both NFAT and FKHR transcriptional activity in brain ischemia.
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Decreased calcium/calmodulin-dependent protein kinase II and protein kinase C activities mediate impairment of hippocampal long-term potentiation in the olfactory bulbectomized mice

Shigeki Moriguchi, +4 more
- 01 Apr 2006 - 
TL;DR: In OBX mice NMDA receptor hypofunction, possibly through decreased PKCα activity, underlies decreased CaMKII activity in the post‐synaptic regions, thereby impairing LTP induction in the hippocampal CA1 region.