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Anna Nogalska

Researcher at University of Southern California

Publications -  47
Citations -  5684

Anna Nogalska is an academic researcher from University of Southern California. The author has contributed to research in topics: White adipose tissue & Inclusion body myositis. The author has an hindex of 22, co-authored 42 publications receiving 4681 citations. Previous affiliations of Anna Nogalska include Gdańsk Medical University.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

p62/SQSTM1 is overexpressed and prominently accumulated in inclusions of sporadic inclusion-body myositis muscle fibers, and can help differentiating it from polymyositis and dermatomyositis

Anna Nogalska, +4 more
- 26 Jun 2009 - 
TL;DR: In s-IBM muscle fibers, p62 protein is increased on both the protein and the mRNA levels, and it is strongly accumulated within, and as a dense peripheral shell surrounding, p-tau containing inclusions, by both the light- and electron-microscopy.
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Inclusion Body Myositis: A Degenerative Muscle Disease Associated with Intra-Muscle Fiber Multi-Protein Aggregates, Proteasome Inhibition, Endoplasmic Reticulum Stress and Decreased Lysosomal Degradation

Valerie Askanas, +2 more
- 01 Jul 2009 - 
TL;DR: Abnormal intracellular accumulation of unfolded proteins may lead to their aggregation and inclusion body formation, and s‐IBM can be considered a "conformational disorder" caused by protein unfolding/misfolding combined with the formation of inclusion bodies.
Journal ArticleDOI

Impaired Autophagy in Sporadic Inclusion-Body Myositis and in Endoplasmic Reticulum Stress-Provoked Cultured Human Muscle Fibers

Anna Nogalska, +4 more
- 01 Sep 2010 - 
TL;DR: In s-IBM muscle, decreased lysosomal proteolytic activity might enhance accumulation of misfolded proteins, despite increased maturation of autophagosomes, and that ERS is a possible cause of s- IBM-impaired lysOSomal function.
Journal ArticleDOI

Sporadic inclusion-body myositis: A degenerative muscle disease associated with aging, impaired muscle protein homeostasis and abnormal mitophagy.

Valerie Askanas, +2 more
- 01 Apr 2015 - 
TL;DR: The intriguing phenotypic similarities between s-IBM muscle fibers and the brains of Alzheimer and Parkinson's disease patients, the two most common neurodegenerative diseases associated with aging, are discussed.