scispace - formally typeset
M

Masashi Narita

Researcher at University of Cambridge

Publications -  115
Citations -  27421

Masashi Narita is an academic researcher from University of Cambridge. The author has contributed to research in topics: Senescence & Autophagy. The author has an hindex of 43, co-authored 95 publications receiving 22729 citations. Previous affiliations of Masashi Narita include Cold Spring Harbor Laboratory & Howard Hughes Medical Institute.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

Shigeomi Shimizu, +2 more
- 03 Jun 1999 - 
TL;DR: The results indicate that the Bcl-2 family of proteins bind to the VDAC in order to regulate the mitochondrial membrane potential and the release of cytochrome c during apoptosis.
Journal ArticleDOI

Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence.

Masashi Narita, +9 more
- 13 Jun 2003 - 
TL;DR: A distinct heterochromatic structure that accumulates in senescent human fibroblasts is described, which is designated senescence-associated heterochROMatic foci (SAHF) and is associated with the stable repression of E2F target genes.
Journal ArticleDOI

Reversal of human cellular senescence: roles of the p53 and p16 pathways

Christian Beauséjour, +8 more
- 15 Aug 2003 - 
TL;DR: The results indicate that the senescence response to telomere dysfunction is reversible and is maintained primarily by p53, however, p16 provides a dominant second barrier to the unlimited growth of human cells.