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Noor Gammoh

Researcher at University of Edinburgh

Publications -  22
Citations -  8377

Noor Gammoh is an academic researcher from University of Edinburgh. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 18, co-authored 20 publications receiving 6887 citations. Previous affiliations of Noor Gammoh include Memorial Sloan Kettering Cancer Center & Edinburgh Cancer Research Centre.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

SNARE proteins are required for macroautophagy

TL;DR: Evidence is provided for the involvement of exocytic Q/t-SNAREs in autophagosome formation, acting in the recruitment of key autophagy components to the site of autophagic formation, and in regulating the organization of Atg9 into tubulovesicular clusters.
Journal ArticleDOI

Distinct autophagosomal-lysosomal fusion mechanism revealed by thapsigargin-induced autophagy arrest.

TL;DR: Molecularly, it is found that Rab7 but not Vps16 is required for complete autophagy flux, and that thapsigargin blocks recruitment of Rab7 to autophagosomes, which means thatAutophagosomal-lysosomal fusion must be governed by a distinct molecular mechanism compared to general endocytic fusion.
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Role of autophagy in histone deacetylase inhibitor-induced apoptotic and nonapoptotic cell death

TL;DR: Autophagy functions as a prosurvival mechanism to mitigate SAHA-induced apoptotic and nonapoptotic cell death, suggesting that targeting autophagy might improve the therapeutic effects of SAHA.