scispace - formally typeset
F

Federico Pietrocola

Researcher at Karolinska Institutet

Publications -  84
Citations -  15320

Federico Pietrocola is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Autophagy & Medicine. The author has an hindex of 36, co-authored 70 publications receiving 11491 citations. Previous affiliations of Federico Pietrocola include Paris Descartes University & Pierre-and-Marie-Curie University.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Acetyl Coenzyme A: A Central Metabolite and Second Messenger

TL;DR: By influencing the acetylation profile of several proteins, including histones, acetyl-CoA controls key cellular processes, including energy metabolism, mitosis, and autophagy, both directly and via the epigenetic regulation of gene expression.
Journal ArticleDOI

Cardioprotection and lifespan extension by the natural polyamine spermidine

Tobias Eisenberg, +69 more
- 01 Dec 2016 - 
TL;DR: It is shown that oral supplementation of the natural polyamine spermidine extends the lifespan of mice and exerts cardioprotective effects, reducing cardiac hypertrophy and preserving diastolic function in old mice, and suggests a new and feasible strategy for protection against cardiovascular disease.