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Joaquín Jordán

Researcher at University of Castilla–La Mancha

Publications -  155
Citations -  14104

Joaquín Jordán is an academic researcher from University of Castilla–La Mancha. The author has contributed to research in topics: Mitochondrion & Programmed cell death. The author has an hindex of 45, co-authored 154 publications receiving 12336 citations. Previous affiliations of Joaquín Jordán include University of Chicago & Universidad Miguel Hernández de Elche.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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The PI 3-kinase/Akt signaling pathway delivers an anti-apoptotic signal.

TL;DR: The results suggest that uncoupling of survival and mitogenesis can be explained by differing abilities of distinct mitogens to efficiently induce the PI 3-kinase/Akt signaling pathway.
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Superoxide production in rat hippocampal neurons: selective imaging with hydroethidine

TL;DR: Digital-imaging microfluorimetry of the oxidation of hydroethidine (HEt) to ethidium can be used to monitor superoxide (O2-) production selectively within individual rat hippocampal pyramidal neurons in culture and in brain slices.
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Mutant Superoxide Dismutase-1-Linked Familial Amyotrophic Lateral Sclerosis: Molecular Mechanisms of Neuronal Death and Protection

TL;DR: The hypothesis that mutant SOD induced-neurodegeneration is associated with disturbances of neuronal free radical homeostasis is supported by the results.