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Alon Kahana

Researcher at University of Michigan

Publications -  103
Citations -  8806

Alon Kahana is an academic researcher from University of Michigan. The author has contributed to research in topics: Zebrafish & Regeneration (biology). The author has an hindex of 25, co-authored 95 publications receiving 7222 citations. Previous affiliations of Alon Kahana include University of Chicago & University of Illinois at Chicago.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Identification of High-Copy Disruptors of Telomeric Silencing in Saccharomyces cerevisiae

TL;DR: A genetic screen to identify genes whose overexpression disrupts telomeric silencing isolated 10 DOT genes, which include both components and regulators of silent chromatin, suggesting that DOT1 and DOT4 normally play important roles in gene repression.
Journal ArticleDOI

Intravitreal triamcinolone for the treatment of macular edema associated with central retinal vein occlusion.

TL;DR: Intravitreal injection of triamcinolone appears to be a possibly effective treatment in some patients with macular edema associated with central retinal vein occlusion and patients with nonischemic CRVO may respond more favorably than patients with ischemic CRVO.
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The ubiquitin-conjugating enzyme Rad6 (Ubc2) is required for silencing in Saccharomyces cerevisiae.

TL;DR: It is shown that a null mutation of the DNA repair gene RAD6 reduces silencing of the HM loci and lowers the mating efficiency of MATa strains, indicating that the ubiquitin-conjugating activity of RAD6 is essential for full telomeric silencing.