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Joan Villarroya

Researcher at University of Barcelona

Publications -  69
Citations -  9189

Joan Villarroya is an academic researcher from University of Barcelona. The author has contributed to research in topics: Adipose tissue & Brown adipose tissue. The author has an hindex of 24, co-authored 62 publications receiving 7132 citations. Previous affiliations of Joan Villarroya include Albert Einstein College of Medicine & University of Santiago de Compostela.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Brown adipose tissue as a secretory organ

Francesc Villarroya, +3 more
- 01 Jan 2017 - 
TL;DR: The current understanding of the regulatory molecules that are released by BAT that influence systemic metabolism and convey the beneficial metabolic effects of BAT activation are discussed.
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An endocrine role for brown adipose tissue

Joan Villarroya, +2 more
- 01 Sep 2013 - 
TL;DR: Whether brown fat plays an endocrine role and, if so, to comprehensively identify which endocrine factors are released by BAT is investigated.
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Inflammation of brown/beige adipose tissues in obesity and metabolic disease

Francesc Villarroya, +4 more
- 01 Nov 2018 - 
TL;DR: The targeting of brown and beige adipose tissues by pro‐inflammatory signals and the subsequent impairment of their thermogenic and metabolite draining activities appears to represent obesity‐driven disturbances that contribute to metabolic syndrome and cardiovascular alterations in obesity.