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Akiko Maeda

Researcher at Case Western Reserve University

Publications -  104
Citations -  11728

Akiko Maeda is an academic researcher from Case Western Reserve University. The author has contributed to research in topics: Retinal & Retinal degeneration. The author has an hindex of 44, co-authored 101 publications receiving 9887 citations. Previous affiliations of Akiko Maeda include University of Washington & University of California, Irvine.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Essential role of Ca2+-binding protein 4, a Cav1.4 channel regulator, in photoreceptor synaptic function.

TL;DR: Observations indicate that CaBP4 is important for normal synaptic function, probably through regulation of Ca2+ influx and neurotransmitter release in photoreceptor synaptic terminals.
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Retinopathy in Mice Induced by Disrupted All-trans-retinal Clearance

TL;DR: These findings provide direct evidence that aberrant production of toxic condensation byproducts of the visual cycle in mice can lead to rapid, progressive retinopathy and severe RPE/photoreceptor dystrophy at an early age.
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Involvement of All-trans-retinal in Acute Light-induced Retinopathy of Mice

TL;DR: Results indicate that this acute light-induced retinopathy requires the presence of free all-trans-retinal and not, as generally believed, A2E or other retinoid condensation products, which further understanding of the mechanisms involved in light- induced retinal degeneration.