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Mar Lorente

Researcher at Complutense University of Madrid

Publications -  57
Citations -  10132

Mar Lorente is an academic researcher from Complutense University of Madrid. The author has contributed to research in topics: Cannabinoid & Cancer. The author has an hindex of 29, co-authored 54 publications receiving 8292 citations. Previous affiliations of Mar Lorente include Spanish National Research Council & Salisbury University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Cannabinoids Induce Apoptosis of Pancreatic Tumor Cells via Endoplasmic Reticulum Stress–Related Genes

TL;DR: Results presented here show that cannabinoids lead to apoptosis of pancreatic tumor cells via a CB(2) receptor and de novo synthesized ceramide-dependent up-regulation of p8 and the endoplasmic reticulum stress-related genes ATF-4 and TRB3.
Journal ArticleDOI

The stress-regulated protein p8 mediates cannabinoid-induced apoptosis of tumor cells

TL;DR: The stress-regulated protein p8 is identified as an essential mediator of cannabinoid antitumoral action and it is shown that p8 upregulation is dependent on de novo-synthesized ceramide and mediates its apoptotic effect via upregulation of the endoplasmic reticulum stress-related genes ATF-4, CHOP, and TRB3.