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Craig Stevens

Researcher at Edinburgh Napier University

Publications -  65
Citations -  9071

Craig Stevens is an academic researcher from Edinburgh Napier University. The author has contributed to research in topics: Autophagy & Medicine. The author has an hindex of 28, co-authored 58 publications receiving 7496 citations. Previous affiliations of Craig Stevens include Western General Hospital & University of Glasgow.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Chk2 activates E2F-1 in response to DNA damage

Craig Stevens, +2 more
- 01 May 2003 - 
TL;DR: It is reported that checkpoint kinase 2 (Chk2) regulates E2F-1 activity in response to the DNA-damaging agent etoposide, resulting in apoptosis and a role for E2f-1 in checkpoint control and a plausible explanation for the tumour suppressor activity of E2 F-1.
Journal ArticleDOI

E2F and cell cycle control: a double-edged sword.

Craig Stevens, +1 more
- 15 Apr 2003 - 
TL;DR: E2F appears to play a pivotal role in coordinating events connected with proliferation, cell cycle arrest, and apoptosis, and in regulating cellular proliferation through its temporal association with pocket protein family members.
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Autophagic targeting of Src promotes cancer cell survival following reduced FAK signalling

Emma Sandilands, +13 more
- 01 Jan 2012 - 
TL;DR: C-Cbl-mediated autophagic targeting of active Src can occur in cancer cells to maintain viability when flux through the integrin/Src/FAK pathway is disrupted, exposing a previously unrecognized cancer cell vulnerability that may provide a new therapeutic opportunity.