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Masaki Tanaka

Researcher at Kyoto Prefectural University of Medicine

Publications -  223
Citations -  12283

Masaki Tanaka is an academic researcher from Kyoto Prefectural University of Medicine. The author has contributed to research in topics: Internal medicine & Hypothalamus. The author has an hindex of 38, co-authored 201 publications receiving 10349 citations. Previous affiliations of Masaki Tanaka include Fujita Health University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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New neuropeptides containing carboxy-terminal RFamide and their receptor in mammals.

Shuji Hinuma, +21 more
- 01 Oct 2000 - 
TL;DR: In this paper, the identification of a human gene that encodes at least three RFamide-related peptides, hRFRP-1-3, has been reported.
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Contributions of high mobility group box protein in experimental and clinical acute lung injury

Hiroshi Ueno, +12 more
- 15 Dec 2004 - 
TL;DR: Examination of the putative role of high mobility group box (HMGB) protein in the pathogenesis of acute lung injury suggests that it also plays a physiologic role in the lung and extracellular HMGB1 expression in normal airways is noteworthy and suggests this protein may play a key role in clinical and experimental ALI.
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NGF and GDNF differentially regulate TRPV1 expression that contributes to development of inflammatory thermal hyperalgesia

Fumimasa Amaya, +7 more
- 01 Nov 2004 - 
TL;DR: It is suggested that inflammation differentially increases both NGF and GDNF, which facilitate TRPV1 expression within distinctive neurons to induce thermal hyperalgesia.